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With Friedreich's ataxia, there is a disorder of zantac system, which can manifest itselfin the form of the following diseases: ovarian dysfunction; diabetes; infantilism; hypogonadism.

Very often in patients with Friedreich's ataxia, a cataract is found, so it is also considered part of the clinical picture of this disease. Friedreich's ataxia is characterized by rapid progression and an increase in symptoms. The duration of the disease is often no more than twenty years. A pronounced clinical picture of Friedreich's atypical ataxia is observed later than in the classical form - approximately in the third to fifth decade of a person's life.

The course occurs in a lighter form than with classical ataxia and the outcome of the disease is more favorable: The patient retains the ability to self-service for a long time. There is no development of ranitidine. No paresis, reflexes are preserved. Diagnosis of Friedreich's ataxia.

Diagnosis of this genetic disease is difficult. Extraneural symptoms without neurological signs often complicate diagnosis. Concomitant diseases (diabetes mellitus, cardiomyopathy, etc.) are treated as individual diseases, and not signs of Friedreich's ataxia.

The lack of adequate treatment accelerates the progression of ranitidine and leads it to a severe stage.

The main diagnostic method of all ataxias is considered to be computed tomography of the brain. But in this case, it is ineffective, since most of the changes in the brain in Friedreich's ataxia are detected only in the later stages. This is due to the spinal localization of changes. The early stages of the disease are not visible on CT.

Conceived in the later stages, only slight atrophy of the cerebellum and hemispheres, some expansion of the cerebral cisterns, lateral ventricles, and subarachnoid space can be detected. An MRI scan is prescribed, with the help of which it is possible to detect atrophy in the spinal cord in the early stages, and the transverse dimensions of zantac pills cord are also examined. With Friedreich's ataxia, they are below normal. Moderate atrophy of the pons, cerebellum and medulla oblongata is also visible.

With the help of an electrophysiological study, the degree of damage to the sensitivity of the nerves of the limbs is established. With Friedreich's ataxia, the amplitude of the action potentials of the sensitivity of the nerves of the extremities is significantly reduced or completely absent.

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Laboratory tests are also prescribed - a blood test to detect glucose tolerance. This analysis is carried out as an opportunity to exclude or confirm one of the concomitant diseases - diabetes mellitus. A laboratory test for the study of hormones is carried out. X-ray examination of zantac 300mg-150mg is prescribed.

The most important method for diagnosing Friedreich's ataxia is DNA diagnostics. To do this, the patient's blood samples are compared with those of both parents and immediate blood relatives. This disease can be detected in the fetus as early as 8-12 weeks of fetal development. DNA diagnostics of chorionic villi is also carried out. Sometimes amniotic fluid is taken from the fetus to detect this disease (at 16-24 weeks).

Diagnosis of this genetic disease is not an easy process due to the difficulty of differentiating the disease in a number of other, almost identical, and often concomitant diseases: Hereditary ataxia due to vitamin E deficiency. For differentiation, the concentration of vitamin E in the blood is determined, the lipid profile of ranitidine pills is examined, and the presence of acanthocytosis is detected using a blood smear. Bassen-Kornzweig syndrome. Diseases associated with metabolic disorders that are inherited in an autosomal recessive manner. For example, Krabbe disease, Niemann-Pick disease. Multiple sclerosis.